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Universidad de Ciencias Aplicadas y Ambientales

Química Farmacéutica · 2019

Estudio de efecto vasorelajante del extracto etanólico Campanula medium aislamiento biodirigido del compuesto activo y posible mecanismo de acción

Núñez Márquez, Jeidy Marlin · Escobar Romero, Yandi YashanaAsesor: Estrada Semprun, Omar, dir. · Urrego Novoa, José Ricardo, dir.

Cardiovascular disease is a health problem worldwide. In Colombia, there is a 45% prevalence of hypertension, associated complications such as heart failure, coronary ischemia multifactorial where the risk factor is one of the highest of Latin American countries. The use of pharmacological treatments has been shown to reduce morbidity and mortality from cardiovascular disease, reducing complications associated with hypertension. For this reason today the search for new alternatives continues with antihypertensive potential; in these order natural products as a source for obtaining biological active molecules are explored. Previous studies have shown that members of the bellflower family have potentially beneficial cardiovascular level activities with antioxidant and / or anti-inflammatory. Therefore the aim of this study was to evaluate the vasorelaxant effect of medium C. in an assay by bioassay-guided technique isolated organ in rat aortic rings, characterize the compound of greater activity and suggest a possible mechanism of action.(He et al., 2014) The results showed that the hydroalcoholic extract of Campanula medium CmXO had two compounds vasorelaxant effect endothelium-dependent. Solubility by a method of solvent and column chromatography and Sephadex LH-20 reverse phase RP18 was achieved isolate two compounds; an already known and reported in the literature as routine and a diterpene named by us as CmE1B, the latter the largest vasorelaxant activity. This compound was characterized qualitatively by thin layer chromatography and then analyzed by NMR and H-1 and C-13 NMR was determined that a diterpene type labdane dihydroxyl. By isolated organ technique it determined that CmE1B has an endothelium-dependent vasorelaxant activity, acting primarily via the nitric oxide and cGMP and through the activation of K + channels dependent Ca2 + in endothelial cells

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